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3Zhao KS (2018) Fifty years in shock research. In: Zhao KS et al eds, "Advances in the Mechanism of Cardio-Celebral Disease", Research Signpost: Kerala (India) 1-40.
4Jin CH, Zhao KS (2009) The effects and mechanism of polydatin in shock treatment, In Zhao KS, Xu Q eds. "Molecular Mechanism of Severe Shock", Research Signpost. Kerala 192-223.
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8Morishima I, sone T, Okumura K, Tsnboi H, Kondo J, et al. (2000) Angiographic no-reflow phenomenon as a predictor of adverse low term outcome in patients treated with percutaneous transluminal coronary angioplasty for first acute myocardial infarction. J Amer College Cardiol 30:1202-1209.
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11Intaglietta M (1999) Microcirculatory basis for the design of artificial blood. Microcirculation 6:247-258.
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15Wu X, Huang X, Zhao KS (1996) Effect of CD11a-CD18 monoclonal antibody on the microcirculation disorder of rabbits in endotoxic shock. Med Coll PLA 11:151-154.
16Zhao K (1996) Advances in the study on rheological behavior of leukocyte during severe shock. Chin Med J 109 : 110-111.
17Zhu Z, Zhao KS (1994) Effects of crystal NO4 of Polygonum cuspidatum on the systemic circulation in rabbits and microcirculation in rat during severe hemorrhagic shock. In: Zhao KS, et al. (eds) "Advances in Burns and Trauma" International Academic Publishers: Beijing 63-68.
18Zhao KS, Wu X, Wang Y (1994) Effect of Polygonum Cuspidatum on the leukocyte activation and adhesion of rat during burn shock. In Niimi H, et al, (eds) "Progress in Microcirculation Research", Elsevier Science Ltd: Oxford 55-58.
19Zhao KS, Woo GY, Tian Y, Zhu ZG (1985) The effect of Polygonum cuspidatum on the microcirculation of burned skin. In:Chang HM (eds) "Advances in Chinese Medicinal Material Research", World Scientific Publ: Singapore 591-595.
20Zhao KS, Zhu Z, Wu K, Huang X (1990) Effect of crystal No4 of polygonum cuspidatum on cardio function of rats with hemorrhagic shock. J Med Coll PLA 5: 12-16.
21Zhao KS, Zhu ZJ, Wu KY, Huang XL (1991) Importence of enhancing cardiac function in the treatment of burn shock - Effect of crystal No4 of Polygounm cuspidate. In: Sheng C, Zhu Z, eds. "Advances in Burns", Interl Acad Pub: Beijing 31-38.
22Zhao K, Liu J, Jin C (2000) The role of membrane potential and calcium kinetic changes in the pathogenesis of vascular hyporeactivity during severe shock. Chin Med J 113:59-64.
23Zhao Q, Zhao KS (2007) Inhibition of L-type calcium channels in arteriolar smooth muscle cells is involved in the pathogenesis of vascular hyporeactivity in severe shock. Shock 28 :717-721.
24Zhao G, Zhao Y, Pan B, Liu J, Huang X, et al. (2007) Hypersensitivity of BKCa to Ca2+ sparks underlies hyporeactivity of arterial smooth muscle in shock. Circ Res. 31:493-502.
25Pan BX, Zhao GL, Huang XL, Jin JQ, Zhao KS (2004) Peroxynitrite induces arteriolar smooth muscle cells membrane hyperpolarization with arteriolar hyporeactivity in rats. Life Sci 74:1199-1210.
26Pan BX, Zhao GL, Huang XL, Zhao KS (2004) Calcium mobilization is required for peroxynitrite-mediated enhancement of spontaneous transient outward currents in arteriolar smooth muscle cells. Free Radic Biol Med. 37:823-838.
27Pan BX, Zhao GL, Huang XL, Zhao KS (2004) Mobilization of intracellular calcium by peroxynitrite in arteriolar smooth muscle cells from rats. Redox Rep 9:49-55.
28Liu J, Zhao KS (2000) The ATP-sensitive K+ channel and membrane potential in the pathogenesis of vascular hyporeactivity in severe hemorrhagic shock. Chin J Trauma 3:39-44.
29Liu J, Zhao K.S, Jin CH, Huang QB (1999) Effect of intracellular acidosis on the pathogenesis of vascular hyporeactivity in severe hemorrhagic shock. Crit Care Shock 3:112-118.
30Zhao KS, Huang X, Liu J, Huang Q, Jin C, et al. (2002) New approach to treatment of shock-restitution of vasoreactivity. Shock 18:189-192.
31Song R, Bian H, Wang X, Huang X, Zhao KS (2011) Mitochondrial injury underlies hyporeactivity of arterial smooth muscle in severe shock. Am J Hypertens 24:45-51.
32Wang X, Song R, Bian HN, Brunk UT, Zhao M, et al. (2012) a natural polyphenol, protects arterial smooth muscle cells against mitochondrial dysfunction and lysosomal destabilization following hemorrhagic shock. Am J Physiol Regul Integr Comp Physiol 302 : 805-814.
33Wang X, Song R, Chen Y, Zhao M, Zhao KS (2013) Polydatin--a new mitochondria protector for acute severe hemorrhagic shock treatment. Expert Opin Investig Drugs 22:169-179.Wang X, Song R, Chen Y, Zhao M, Zhao KS (2013) Polydatin--a new mitochondria protector for acute severe hemorrhagic shock treatment. Expert Opin Investig Drugs 22:169-179.
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35Zeng Z, Chen Z, Xu S, Zhang Q, Wang X, Gao Y, Zhao KS (2016) Polydatin protecting kidneys against hemorrhagic shock-induced mitochondrial dysfunction via SIRT1 activation and p53 deacetylation. Oxid Med Cell Longev. 1737185. 1-16.
36Zhao KS, Song R, Wang XM (2014) Mitochondrial dysfunction in severe hemorrhagic shock. Advances in Medicine and Biology 77: 119-133.
37Zheng ZH, Xu SQ, Zhao KS (2017) Protection against mitochondrial and cell damage a new approach to treat severe hemorrhagic shock. Advances in Medicine Biology 110: 61- 93.
38Chen YY, Wang XM, Song R, Zhao KS (2012) Changes of platelet mitochondria in rats with severe hemorrhagic shock and intervention effect of polydatin. Chin J Trauma 29: 822-889.
39Li P, Liu Y, Burns N, Zhao KS, Song R (2017) SIRT1 is required for mitochondrial biogenesis reprogramming in hypoxic human pulmonary arteriolar smooth muscle cells. Inter J Mol Med 39:1127-1136.
40Li P, Wang X, Zhao M, Song R, Zhao KS (2015) Polydatin protects hepatocytes against mitochondrial injury in acute severe hemorrhagic shock via SIRT1- SOD2 pathway. Expert Opin Ther Targets 19 : 997-1010.
41Meng X, Tan J, Li M, Song S, Miao Y, Zhang Q (2017) Sirt1: role under the condition of ischemia/hypoxia. Cell Mol Neurobiol 37:17-28.
42Siqi Xu, Youguang Gao, Qin Zhang, Siwei Wei, et al. (2016) SIRT1/3 activation by resveratrol attenuates acute kidney injury in a septic rat model. Oxidative Medicine and Cellular Longevity Volume Article. 2016, 7296092. 1-12.
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44Yang H, Gu ZT, Li L, Maegele M, Zhou BY, et al. (2017) SIRT1 plays a neuroprotective role in traumatic brain injury in rats via inhibiting the p38 MAPK pathway. Acta Pharmacol Sin 38:168-181.
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